The Distinguished Faculty Research Award is presented annually to a faculty member whose research has made an impact on a broad field of science by contributing to the solution of a significant scientific problem, or whose work shows ingenuity and originality in the application of novel techniques to an important area of science. This year we are honoring:

R. Stephen Lloyd received his B.S. from Florida State University, Tallahassee, in 1975, and his Ph.D. from the University of Texas Graduate School of Biomedical Sciences, Houston, in 1979.  He completed a postdoctoral fellowship at Stanford University in 1981, prior to accepting a position as senior research scientist and Genex Corporation in Gaithersburg, Maryland.  Dr. Lloyd served on the faculty at Vanderbilt University, School of Medicine from 1988 to 1992, where he was professor of biochemistry, director of the molecular genetics core and director of the cell biology and immunology core. He joined the faculty at UTMB in 1992. 

Dr. Lloyd holds the Mary Gibbs Jones Distinguished Chair in Environmental Toxicology and is director of the National Institute of Environmental Health Sciences Center, and director of the Sealy Center for Environmental Health and Medicine and is professor in the department of Biochemistry and Molecular Biology.  

R. Stephen Lloyd, Ph.D.
Professor
Human Biological Chemistry 
and Genetics

Dr. Lloyd’s area of research is DNA damage and repair.  His recent results on MutY, a major DNA repair enzyme responsible for the removal of oxidatively damaged DNA, are seminal including fundamental enzymology and structural biology. He has also developed methods that may prove important in the repair of UV-damaged DNA, which can result in skin cancer. According to a letter of nomination, investigations focused on DNA-protein crosslink (DPC) have been hindered due to technical constraints.  However, Dr. Lloyd and his co-workers developed methodology, which provides a valuable tool in the development of a number of assays that will aid in the elucidation of the cellular response to a largely unexplored class of DNA damage. This methodology is amenable to studying the mutagenic and carcinogenic potential of DPC lesions, thus providing mechanistic insight linking DPC damaged to human health and disease. 

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